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Possible biologic mechanisms for exercise-induced reductions in all-cause and cardiac mortality

Possible biologic mechanisms for exercise-induced reductions in all-cause and cardiac mortality
Cardiovascular influences
Reduction of resting and exercise heart rate
Reduction of resting and exercise blood pressure
Reduction of myocardial oxygen demand at submaximal levels of physical activity
Expansion of plasma volume
Increase in myocardial contractility
Increase in peripheral venous tone
Favorable changes in fibrinolytic system
Increased endothelium-dependent vasodilation
Increased gene expression for nitric oxide synthase
Enhanced parasympathetic tone
Possible increases in coronary blood flow, coronary collateral vessels, and myocardial capillary density
Metabolic influences
Reduction of obesity
Enhanced glucose tolerance
Improved lipid profile
Lifestyle influences
Decreased liklihood of smoking
Possible reduction of stress
Short-term reduction of appetite
Mechanism_exercise_on_morta.htm
Data from: Shepard RJ, Balady GJ. Circulation 1999; 99:963.
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